慢性骨髓白血病；慢性粒細胞白血病
Front-Line Therapy With Second-Generation Tyrosine Kinase Inhibitors in Patients With Early Chronic Phase Chronic Myeloid Leukemia: What Is the Optimal Response?
使用二代酪氨酸激酶抑制劑作為一線療法治療早期慢性期慢性粒細胞白血病患者：什麼是最佳的反應？
Recently, elevated IL-18 levels were found in serum from some leukemia patients, especially those with acute lymphocytic leukemia (ALL) and chronic myeloid leukemia (CML).
近來，研究發現急性淋巴細胞白血病（ALL）和慢性粒細胞白血病（CML）患者血清中IL-18水平高于正常人。
Objective To analyze the frequency and clinical significance of ABL tyrosine kinase point mutations in chronic myeloid leukemia (CML) patients receiving imatinib treatment.
目的探讨伊馬替尼治療慢性粒細胞白血病（CML）患者ABL酪氨酸激酶區點突變的發生情況及臨床意義。
Objective To study the gene expression profile of chronic myeloid leukemia(CML) in chronic and blast phases for providing information about the potential mechanisms of blastic crisis.
目的研究慢性粒細胞白血病（CML）不同臨床階段的基因表達差異，為闡明CML演變的可能機制提供依據。
Eight samples of bone marrow coming from patients affected by acute myeloid leukemia (AML) and nine samples from patients suffered from chronic myeloid leukemia(CML) were stu***d by FTIR spectroscopy.
研究了急性粒細胞白血病（ AML）和慢性粒細胞白血病（ CML）患者的骨髓淋巴細胞、粒細胞和紅細胞的傅立葉紅外光譜（ FTIR） 。
Objective To evaluate the prognostic factors of chronic myeloid leukemia(CML).
目的研究影響慢性粒細胞白血病（CML）生存的預後因素。
Philadelphia chromosome formed may result in the generation of the fusion protein BCR ABL, a constitutively activated tyrosine ki nas e which then causes chronic myeloid leukemia (CML).
慢性粒細胞白血病（CML）患者體内有異常染色體形成并産生BCRABL融合蛋白，該融合蛋白能激活酪氨酸激酶，刺激細胞過度增生導緻慢性粒細胞白血病的發生。
Objective: To induce and differentiate into dendritic cells from leukemic cells of patients with chronic myeloid leukemia.
目的：将慢性髓性白血病患者的白血病細胞體外誘導分化成為樹突狀細胞。
A writer and translator living in Manhattan, she was suffering from chronic myeloid leukemia, an especially deadly form of blood cancer.
她是一位作家和翻譯家，住在曼哈頓，當時正患慢性髓細胞樣白血病，一種特殊的緻死性血癌。
Objective:Observation of bone marrow smears of typical chronic myeloid leukemia to investigate the Possibility of morphological classification of the disease.
目的：觀察典型慢性髓系白血病骨髓塗片，探讨用常規血細胞形态學方法分型的可能性。
Objective To detect the ABL point mutations in chronic myeloid leukemia(CML)patients treated with imatinib by denaturing high-performance liquid chromatography(DHPLC).
目的應用變性高效液相色譜（DHPLC）檢測伊馬替尼治療後慢性髓性白血病（CML）患者ABL激酶區點突變。
Objective To explore the value of interphase fluorescence in situ hybridization(I-FISH) on the detection of trisomy 8 in chronic myeloid leukemia(CML) and the progression of CML.
目的探讨間期熒光原位雜交（I-FISH）技術檢測慢性髓系白血病（CML）8號染色體三體（8三體）異常的價值及其對CML演進監測的意義。
Selecting optimal second-line tyrosine kinase inhibitor therapy for chronic myeloid leukemia patients after imatinib failure: does the BCR-ABL mutation status really matter?
選擇最佳的二線酪氨酸激酶抑制劑治療伊馬替尼治療失敗的慢性粒細胞白細胞患者：BCR-ABL突變狀态是否真的重要？
Objective: TO observe the effect of Zubianling capsule in treating chronic myeloid leukemia.
目的觀察阻變靈膠囊治療慢性粒細胞白血病的療效。
Recent research shows that, HHT can be used for the chronic myeloid leukemia patients after the failure of interferon therapy or imatinib-resistant.
近期研究表明，HHT還可用于幹擾素治療失敗和伊馬替尼耐藥的慢性粒細胞白血病的治療。
Imatinib is currently the most popular therapy for chronic myeloid leukemia (CML).
伊馬替尼是目前治療慢性髓細胞白血病的最普遍藥物。
Objective To study the changes of cytogenetics and molecular genetics in chronic myeloid leukemia blast crisis(CML-BC).
目的研究慢性髓細胞白血病急變期（CML-BC）細胞遺傳學及分子遺傳學改變。
Objective To study the vascular endothelium growth factor (VEGF) antisense oligonucleotide (ASON) Cinobufotalin in enhancing apoptosis induced by human chronic myeloid leukemia K562 cell line.
目的研究VEGF反義核酸（ASON）增強華蟾素對人慢性粒細胞白血病K562細胞株誘導凋亡效應。
All-time NBA top scorer Kareem Abdul-Jabbar has been diagnosed with chronic myeloid leukemia, he said during an interview with the Los Angeles Times.
NBA史上得分王卡裡姆·阿蔔杜爾-賈巴爾日前在接受《洛杉矶時報》的采訪時披露，自己被确診患上慢性粒細胞性白血病。
Plasma adiponectin levels are markedly elevated in imatinib-treated chronic myeloid leukemia (CML) patients: a mechanism for improved insulin sensitivity in type 2 diabetic CML patients?
用伊馬替尼治療的慢性粒細胞白血病（CML）患者血漿脂聯素濃度顯著升高：這是改善2型糖尿病CML患者胰島素敏感性的一種機制？
Objective To explore the expression of PTTG gene of chronic myeloid leukemia(CML)in different phases for providing information about the potential mechanisms of CML.
目的研究垂體瘤轉化基因（PTTG）在慢性粒性細胞白血病（CML）患者中不同臨床階段的基因表達，為闡明CML演變的可能機制提供依據。
Conclusion OD-PTD fusion protein can significantly inhibit the proliferation of K562 cells, which provides an experimental basis for new therapy of chronic myeloid leukemia.
結論OD-PTD融合蛋白對K562細胞有明顯的抑制作用，為進一步探讨慢性粒細胞白血病新的治療手段提供了實驗依據。
The WT1 expression level of chronic myeloid leukemia(CML) in blast crisis was higher than that of CML in chronic phase.
慢性髓性白血病（CML）急變期患者WT1基因表達水平顯著高于慢性期。
Chronic myeloid leukemia (CML) is a clonal hematologic malignancy that arise in the stem cell compartment .
研究背景慢性髄細胞白血病（CML）是一種起源于多能造血幹細胞的克隆性惡性血液病。
For example, patients with chronic myeloid leukemia, a blood cancer, nearly always have swapped pieces of chromosomes 9 and 22 in their cancer cells.
例如，慢性粒細胞白血病（chronic myeloid leukemia）是一種血細胞癌症，其癌細胞中總會發生9號染色體與22號染色體之間部分的交換。
There is another convincing case related to chronic myeloid leukemia.
另一個令人信服的案例就是慢性骨髓性白血病。
慢性髓系白血病（Chronic Myeloid Leukemia, CML）是一種造血幹細胞惡性克隆性疾病，屬于慢性白血病的一種。以下是詳細解釋：
CML是一種以髓系細胞異常增殖為主的血液系統惡性腫瘤，主要累及粒細胞系。其核心特征是存在Ph染色體（費城染色體）或BCR-ABL融合基因，這是診斷的必要條件。患者外周血中粒細胞顯著增多，并伴隨脾髒腫大。
CML起源于多能造血幹細胞的基因突變，95%以上病例由t(9;22)(q34;q11)染色體易位導緻，形成BCR-ABL融合基因。該基因編碼的異常蛋白具有持續酪氨酸激酶活性，促使細胞不受控增殖。
CML也被稱為慢性粒細胞白血病（Chronic Granulocytic Leukemia），英文縮寫均為CML。其屬于慢性白血病範疇，與急性白血病相比進展較緩慢。
如需進一步了解治療或流行病學數據，可參考權威醫學文獻或臨床指南。
慢性髓細胞白血病（chronic myeloid leukemia）是一種造血幹細胞惡性增殖性疾病，其主要特征是骨髓中存在大量成熟和不成熟的髓細胞，同時外周血中也存在大量的白細胞和幼稚細胞。以下是該詞彙的一些解釋和用法。
慢性髓細胞白血病是一種由骨髓幹細胞引起的血液系統惡性疾病，主要表現為骨髓中存在大量成熟和不成熟的髓細胞，同時外周血中也存在大量的白細胞和幼稚細胞。該疾病常常在成年人中發生，且男性患病率高于女性。治療方法包括藥物治療、幹細胞移植等。
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